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Fut9 Deficiency Causes Abnormal Neural Development in the Mouse Cerebral Cortex and Retina | SpringerLink
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An integrated computational and experimental study uncovers FUT9 as a metabolic driver of colorectal cancer | Molecular Systems Biology
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Cancers | Free Full-Text | FUT9-Driven Programming of Colon Cancer Cells towards a Stem Cell-Like State
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FUT9-specific transcriptional programming of cancer stemness in primary... | Download Scientific Diagram
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PDF] FUT9-Driven Programming of Colon Cancer Cells towards a Stem Cell-Like State | Semantic Scholar
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PDF] FUT9-Driven Programming of Colon Cancer Cells towards a Stem Cell-Like State | Semantic Scholar
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Silencing α1,3-Fucosyltransferases in Human Leukocytes Reveals a Role for FUT9 Enzyme during E-selectin-mediated Cell Adhesion - ScienceDirect
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Structural basis for Lewis antigen synthesis by the α1,3-fucosyltransferase FUT9 | Nature Chemical Biology
Large-Scale Identification of N-Glycan Glycoproteins Carrying Lewis x and Site-Specific N-Glycan Alterations in Fut9 Knockout Mice | Journal of Proteome Research
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Large-Scale Identification of N-Glycan Glycoproteins Carrying Lewis x and Site-Specific N-Glycan Alterations in Fut9 Knockout Mice | Journal of Proteome Research
![α1,3-Fucosyltransferase 9 (FUT9; Fuc-TIX) preferentially fucosylates the distal GlcNAc residue of polylactosamine chain while the other four α1,3FUT members preferentially fucosylate the inner GlcNAc residue - ScienceDirect α1,3-Fucosyltransferase 9 (FUT9; Fuc-TIX) preferentially fucosylates the distal GlcNAc residue of polylactosamine chain while the other four α1,3FUT members preferentially fucosylate the inner GlcNAc residue - ScienceDirect](https://ars.els-cdn.com/content/image/1-s2.0-S0014579399015495-gr4.gif)
α1,3-Fucosyltransferase 9 (FUT9; Fuc-TIX) preferentially fucosylates the distal GlcNAc residue of polylactosamine chain while the other four α1,3FUT members preferentially fucosylate the inner GlcNAc residue - ScienceDirect
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Discovery of regulatory code for glycosylation: Expectations for the development of biopharmaceuticals | News | Science Japan
![Fut9 Deficiency Causes Abnormal Neural Development in the Mouse Cerebral Cortex and Retina | SpringerLink Fut9 Deficiency Causes Abnormal Neural Development in the Mouse Cerebral Cortex and Retina | SpringerLink](https://media.springernature.com/lw685/springer-static/image/art%3A10.1007%2Fs11064-022-03651-8/MediaObjects/11064_2022_3651_Fig1_HTML.png)
Fut9 Deficiency Causes Abnormal Neural Development in the Mouse Cerebral Cortex and Retina | SpringerLink
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Probing substrate glycans of FUT2, FUT6, FUT7 and FUT9 on fetuin (Fet)... | Download Scientific Diagram
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